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Immunological Effects of Low-Fat Diets with and without Weight Loss

Nutritional Immunology Laboratory (M.S.S., L.S.L., S.N.M.) Jean Mayer USDA Human Nutrition Research Center on Aging, Tufts University
Lipid Laboratory (A.H.L., E.J.S.) Jean Mayer USDA Human Nutrition Research Center on Aging, Tufts University
Department of Pathology, Sackler Graduate School of Biomedical Sciences (S.N.M.)Tufts University, Boston, Massachusetts
Department of Community Medicine, School of Medicine (B.G.) Tufts University, Boston, Massachusetts
School of Family Ecology and Nutrition (M.S.S.) University of Puerto Rico at Rio Piedras, San Juan, Puerto Rico

View larger version (31K): [in a new window]   Fig. 1. Maximum delayed-type hypersensitivity skin responses after BL (baseline, 35 E% fat), RF (reduced-fat, 29 E% fat, isocaloric), LF (low-fat, 15 E% fat, isocaloric) and LFCR (low-fat, 15 E% fat, voluntarily calorie restricted) diets (Mean ± SEM; n = 7). *Significantly greater than baseline, p = 0.005 (paired t test with Bonferroni adjustments for multiple comparisons).

View larger version (28K): [in a new window]   Fig. 2. Lymphocyte proliferative response to ConA (5 mg/L) and PHA (5 mg/L) after BL (baseline, 35 E% fat), reduced-fat, 29 E% fat (isocaloric), LF (low-fat, 15 E% fat, isocaloric) and LPCR (low-fat, voluntarily calorie restricted) diets. (Mean corrected counts per minute ± SD; n = 7.) *Significantly greater than BL, p < 0.017 (ConA-Wilcoxon signed rank test; PHA-paired t test, both with Bonferroni adjustments for multiple comparisons).